The study also suggests masturbation may also reduce the risk of contracting sexually-transmitted infections.
Masturbation could increase a male primate's chances of having offspring, according to new research.
The act of self-pleasuring may help with successful fertilisation by increasing arousal before sex, the study by University College London said. The scientists also said ejaculating after masturbation helps shed low-quality semen, leaving fresh sperm available for mating.However, the scientists said the benefits of female masturbation - from an evolutionary perspective - remain less clear.Lead researcher Dr Matilda Brindle said:"Our findings help shed light on a very common, but little understood, sexual behaviour and represent a significant advance in our understanding of the functions of masturbation.
Masturbation is common among animals but is thought to be more prevalent in primates, including chimpanzees, apes and humans. The study, published in the journal Proceedings of The Royal Society B, focused on primate animals, both wild and captive.
Brasil Últimas Notícias, Brasil Manchetes
Similar News:Você também pode ler notícias semelhantes a esta que coletamos de outras fontes de notícias.
Man locked down in bank after trying to withdraw £15,000 amid row with staffPolice raced to a HSBC bank in Huddersfield town centre last Friday at around 11.35am over reports of an incident involving a male customer.
Consulte Mais informação »
Tearful Holly Willoughby says she's been 'let down' by Phillip Schofield's lies as she returns to This MorningHolly Willoughby has spoken on air for the first time since Phillip Schofield revealed he had an affair with a young male colleague, saying she feels 'let down' by his lies.
Consulte Mais informação »
Swan left with 'serious' injuries following suspected dog attackThe male bird was put to sleep after suffering a broken wing, broken foot and heavy blood loss
Consulte Mais informação »
Effect of the COVID-19 pandemic on obesity and it is risk factors: a systematic review - BMC Public HealthBackground Coronavirus disease (COVID-19) is a contagious disease caused by the severe acute new coronavirus called SARS-CoV-2. Devastating social, economic, and health service utilisation-related activities. Increased burden and lifestyle changes due to confinement. Objective This study aimed to investigate and determine the determinants of obesity during the coronavirus disease (COVID-19) pandemic from 2019 to 2023. Methods Observational studies published between December 2019 and January 2023 were thoroughly searched using a PRISMA flow chart. PubMed, Google Scholar, Web of Science, HINARI, Scopus, and Embase databases were used. Two reviewers independently identified and critically evaluated the relevant literature. Studies that reported weight gain or involved BMI measurements of 25 kg/m2 or BMI z-scores for children during the COVID-19 lockdown were selected for inclusion. The Newcastle–Ottawa Scale (NOS) was used as a quality assessment instrument in nonrandomised studies to evaluate study quality. All the contributing determinants of weight increase were identified, gathered, and synthesised. Results This systematic review identified 40 studies with a total population of 5,681,813 from 22 countries, of which 74.6% were male. The sample size from included articles ranged from 37 to 5,315,435. Of the 40 selected articles, 24 focused on adults, five on adolescents, three on children, and eight on children and adolescents. Physical inactivity, sedentary behaviour, bad eating habits, behavioural lifestyle, excessive stress, depression, anxiety, behavioural risk factors, sex, and ethnic minorities were associated with obesity during the COVID-19 pandemic lockdown. Conclusion During the COVID-19 pandemic, physical inactivity, sedentary lifestyle, and poor eating patterns were the most common risk factors for obesity. Additionally, unhealthy eating habits, excessive behavioural stress, depression, anxiety, low mood, age, gender, and ethnic minorities have been ident
Consulte Mais informação »
Developmental exposure to polychlorinated biphenyls prevents recovery from noise-induced hearing loss and disrupts the functional organization of the inferior colliculusExposure to combinations of environmental toxins is growing in prevalence, and therefore understanding their interactions is of increasing societal importance. Here, we examined the mechanisms by which two environmental toxins – polychlorinated biphenyls (PCBs) and high-amplitude acoustic noise – interact to produce dysfunction in central auditory processing. PCBs are well-established to impose negative developmental impacts on hearing. However, it is not known if developmental exposure to this ototoxin alters the sensitivity to other ototoxic exposures later in life. Here, male mice were exposed to PCBs in utero, and later as adults were exposed to 45 minutes of high-intensity noise. We then examined the impacts of the two exposures on hearing and the organization of the auditory midbrain using two-photon imaging and analysis of the expression of mediators of oxidative stress. We observed that developmental exposure to PCBs blocked hearing recovery from acoustic trauma. In vivo two-photon imaging of the inferior colliculus revealed that this lack of recovery was associated with disruption of the tonotopic organization and reduction of inhibition in the auditory midbrain. In addition, expression analysis in the inferior colliculus revealed that reduced GABAergic inhibition was more prominent in animals with a lower capacity to mitigate oxidative stress. These data suggest that combined PCBs and noise exposure act nonlinearly to damage hearing and that this damage is associated with synaptic reorganization, and reduced capacity to limit oxidative stress. In addition, this work provides a new paradigm by which to understand nonlinear interactions between combinations of environmental toxins. SIGNIFICANCE STATEMENT: Exposure to common environmental toxins is a large and growing problem in the population. This work provides a new mechanistic understanding of how the pre-and postnatal developmental changes induced by polychlorinated biphenyls could negatively impact the
Consulte Mais informação »