Elephants only rarely develop cancer. That is odd. Cancer, after all, is something of a numbers game: the more cells, the more replications
53 is also in the business of marshalling resources in the service of quality control. When it encounters damaged2. That in turn sets off a chain of events that stimulates a cell to repair any damage. If that fails,p53, then, is a potent anti-cancer agent in the body’s arsenal. But cancer, in many creatures, is a potent adversary. Cancer cells have damaged2 happens without a hitch. The first step in quality control is evaded, and the last steps—repair or self-destruct—are never reached.53.
Peto’s paradox lives on, however. Why have humans not evolved this variegated gang of anti-cancer proteins, or indeed the different suite of cellular protections enjoyed by blue whales, if they would confer such a clear-cut survival benefit? Alas, a common refrain in matters scientific: more research is needed. But that is not to say this work is without potential relevance to humans. Work reported in 2016 revealed that genetically engineering mice to have a few extra copies ofdamage. This latest work suggests something many managers are belatedly finding out: success comes not just with a bigger team, but a more diverse one.
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