Researchers may have found a new biomarker for acute COVID-19 frontiersin
). Taken together, these findings are suggestive of a mechanistic link between IL-26 and severe COVID-19 that deserves further investigation in larger study materials.
It is true that a previous study by Caterino M. et al. failed to detect differences in the serum concentrations of IL-26 among COVID-19 patients with mild, moderate, or severe disease. However, their study did not include healthy control subjects. We think that their uncontrolled approach contributed to the failure to obtain evidence for the involvement of IL-26 in COVID-19. Furthermore, the fact that Caterino M. et al.
In summary, our current pilot study forwards evidence that systemic IL-26 is markedly increased in patients with acute COVID-19, and that it correlates with neutrophil-mobilizing cytokines, a marker of prolonged neutrophil survival, and with markers of tissue damage and hematological alteration, the latter of which are known to signify severe COVID-19.
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