The existence of two viruses at the same time can induce an excess of proteins responsible for the distinctive brain plaques associated with Alzheimer's disease, researchers say.
"But the lab studies suggest that if a new exposure to varicella-zoster virus wakes up dormant herpes simplex virus, they could cause trouble."
There were no increase in the signature Alzheimer's proteins tau and beta-amyloid when the model brain tissue was infected with varicella-zoster alone. However, if the neurons already contained a dormant herpes simplex virus, exposure to the varicella-zoster virus caused the herpes virus to reactivate.
"We know there is a correlation between HSV-1 and Alzheimer's disease, and some suggested involvement of varicella-zoster virus, but what we didn't know is the sequence of events that the viruses create to set the disease in motion," said David Kaplan, a biomedical engineer at Tufts University and the senior author of the paper.
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